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For most of the cancers, it cannot be told which event was the initial cause. However, with molecular biology, it is possible to characterize the mutations within a tumor, and to a certain extent predict its behavior. For example, about half of the tumors are deficient in the tumor suppressor gene p53, also known as "the guardian of the genome". This is associated with poor prospects for the patient, since those tumor cells are unlikely to go into apoptosis (programmed cell death) after they are damaged by therapy. There are more mutations that make a tumor more malignant. Telomerase mutations enable a tumor cell to divide indefinitely. Other mutations enable the tumor to grow new blood vessels to feed it, or to detach from the surrounding tissue, spreading to other parts of the body.
Carcinogenesis (lit.: creation of cancer) is the process of derangement of the rate of cell division.
Treatment of cancer typically involves surgery to remove tumors and nearby lymph nodes to which the cancer may have spread, combined with radiation therapy and/or chemotherapy. The latter two target cells in the body that are rapidly dividing. This includes the cancer cells but also certain healthy ones, which is the reason for the severe side effects of these treatments.
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- Cellscience: A-Z Cancer Directory
Links-based listing in areas such as research, hospitals, charities, funding and education. - OncoLink
Extensive information including news, prevention, clinical trials, conferences and psychosocial support from the University of Pennsylvania Cancer Center. - Cancer Lynx
An online 'zine for patients and professionals. - Conversations In Care
Explores the challenges surrounding physician and patient communications in cancer. - Meds.com: Cancer Glossary
Aims to provide a better understanding of words frequently used in patient care.